ChinesemedicinetreatmentofchroniclivercirrhosisExperienceSeminar[Abstract]modernmedicineforchronicliverdisease,cirrhosis,diseasesresearch,mainlyinthespleensurgicallyremovedthis“temporary”approach,whileinmedicine,“kidneyhomology”ideology,combinedwithmodernmedicineforthediseasemicroscopicunderstanding,forthetreatmentofchronicliverdisease,cirrhosisprovidesanewresearchdirection.[Keywords:]TCMlivercirrhosisLiverdiseaseisacommondiseaseinourcountryandtheworldaboutonetenthofthepopulationarepropertyoftheirpatients,ofwhichthereareveryfewpeoplewithliverdisease,severelivertissuedamageafterliverfibrosisandmakesinternalcirrhosisandeventuallyliverdysfunction,orevenlivercancermaydevelopintoterror.Medicineforliverdiseaseandcirrhosisofliverdiseasecausedbydiseasessuchasresearchforalongtimeareinadilemma,althoughliverdiseasehasbeencommittedtotheiracademicstudies,butsofarhavenot1yetachievedamajorbreakthrough,thetreatmentlevelstillandtwentyorthirtyyearsago.Inourcountry,inavarietyofviraldisseminationundertheinfluenceofalargepartoftheearlychangesinpatientswithlivercirrhosis.Advancedchroniclivercirrhosis,long-termcases,hepatocellulardamage,liverfibrosisafterresults.Causedbythemanydirtyorgandamage,whichismainlyportalhypertensionandliverdysfunction,gastrointestinalbleeding,hepaticencephalopathy,concurrentinfectionsandotherseriouscomplications.shouldthelackoftimelytreatment,themortalityrateisquitehigh.Liverdisease,cirrhosisharmthehumanbodyisverylarge.Liverisaveryimportantorgan,itisnotonlyinvolvedinprotein,clottingfactorsandotherimportantmaterialsynthesis,isalsothebody’s“detoxificationfacility.”Upontheoccurrenceofcirrhosis,itmeansTherearealotofdamagedlivercells,isboundtomakethephysiologicalfunctionofthelivergreatlyreducedif,withfurtheraggravatethecondition,severeliverfunction2disorders,shouldnolongerbetreated,willeventuallydevelopintolivercancer.2pathogenesisoflivercirrhosis2.1Cirrhosisisacommonchronicliverdisease,fromoneormorecausesliverdamage,livershowedaprogressive,diffuse,fibrouslesions.Specificperformanceofawiderangeoflivercelldegenerationandnecrosis,hepatocellularnodularregenerativeandfibroustissuehyperplasiaandnodularregenerationoflivercells,resultinginthenormalhepaticlobulestructurewasdestroyedandbloodcirculationpathwayisconverted,makingtheliverdeformationhardeningandthedevelopmentofcirrhosishadnoclinicallyapparentdiseaseearlysymptoms,itwillbeaseriesoflateliverdysfunction,portalhypertensionandothercomplications.2.2Clinicalmanifestationsofearlystagesofcirrhosisoftheliver,patientswithnoobvioussymptoms,examinationshowedonlymildlyabnormalliverfunctionmaybeatthedevelopmentovertime,thepatient3decreasedappetite,bodyweightloss,lackofbodyweakness,abdominalpain,diarrhea,fever,ascitesandothersymptomsatthistime,ifthelaboratorytestswillbefoundinpatientswithfatandproteinmetabolism.Meanwhile,hardeningofthevariouscomplicationsoccur,thepatienthasaseriousthreat.followingcombinationoftheauthor’sclinicalexperienceandliterature,summarizedthemostcommonclinicalliverdisease,cirrhosistype.2.2.1viralcirrhosislivercirrhosisinChinaaccountedfor40%-60%,mainlybyA,B,C,D,hepatitisEvirus,hepatitisA,andrecentstudiessuggestthatwithoutchronichepatitiswhowithoutcirrhosis,andhepatitisBandCthatiseasytoturnintoachronicchronicactivehepatitisandcirrhosis,hepatitisBisthemostcommonofwhichitandthehepatitisCvirusinducedimmuneabno...