Clinical classification of coronary atherosclerotic heart disease observed and compared care(冠状动脉粥样硬化性心脏病的临床分类观察和比较关心)

Clinicalclassificationofcoronaryatheroscleroticheartdiseaseobservedandcomparedcare1,thecauseanalysis1.1Genderfactors:maleincidenceratewassignificantlyhigherthanwomen,andtheearlyageofonsetisalsorelativelyheavy,theincidenceofwomenthanmenage,andwomenwithcoronaryheartdiseaseincidenceofmenclosetomenopausemayberelatedtohormonelevelsinmenandwomen.1.2Theagefactor:theagefactorincidenceincreasedwithagegraduallyincreasedmenaftertheageof50,womenafterage60isrelativelyfast.1.3factors:theincidenceofmanualworkersengagedinmentalsit-inworkers.1.4Dietaryfactors:overnutrition,calorieintakeistoohigh,andcontainhigheramountsofsaturatedfattyacidsandcholesteroldietcausedincreasedcholesterolandtriglyceridesinthebloodisrecognizedasimportantpathogenicfactor.11.5badhabitsfactors:smokers,andsmokingmorethanahighprevalence,thedifferenceisparticularlyevidentinyoungerpatients.1.6diseasefactors:sufferingfromhighrateofhypertensionandprimaryhyperlipidemia,diabetesandothercontractingthisdisease.1.7Physicalfactors:mentalthanthephysicallaborahighincidenceofobesityisalsomoredeveloped.1.8Geneticfactors:Therearefamilyincidencetendencies.2,theincidenceoftheoreticalanalysis2.1plateletaggregationtheory:Ofatherosclerosiswasinitiallyformedforthedepositionoffibrininplateletaggregationandadhesiononthearterialintima,thrombosis,highbloodlipidsandneurological,psychologicalfactorscancauseexcessivebloodcatecholamines,thuspromotingplateletaggregationandclottingtendencyintheincidenceofplayaroleinhighbloodpressureandbloodcatecholaminesecretion,toomuchcanbecounterproductivetothemechanicaldamageoftheintimalsurface,resultinginthearterialintimaplateletadhesionandthrombusformation.22.2lipidinfiltrationtheory:Thearterialwallitselfcanbesynthesizedlipidsinatheroscleroticplaquelipidismainlyderivedfromplasma,andthisisthelow-densitylipoproteinspenetratethearterialwallwithage,increasedpermeabilityofthearterywall,lipoproteinspenetratetheincreasecausedbyvariousfactorsofhyperlipidemia,bloodviscosity,plaquecloggedcoronaryarterywallofatheroscleroticplaqueformationcausedbythedifferentseverityofmyocardialischemiaandtheformationofcoronaryheartdisease.3,clinicalmanifestations3.1angina:Departmentofmyocardialtransientischemicsymptoms,occurredinthephysicallaborandemotional,thetypicalsymptomsofprecordialsternumdullpainorcrampsparoxysmaloftentotheleftshoulderandleftarmtheinsideoftheupperabdomenradiatingtothepatientpale,fastheartrate,bloodpressure.departmenttreated21patientswithanginapectorisand17,restingsleepspontaneouslycolicsymptomsafteroraladministrationofnitroglycerinvaryingdegreesofrelief.33.2myocardialinfarction(AMI:Departmentofcoronaryarteryiscompletelyclosed,themyocardialbloodsupplytothesharpreductionorinterruptioncausedbyregionalmyocardialischemicnecrosisisaserioustype.Thisdiseaseoccursinmorethanmenovertheageof40,severaldaysbeforetheonsetofaurasymptomssuchasfatigue,chesttightness,shortnessofbreathorangina.Mainly:themajorityofseverechestpainpatientswithsuddenretrosternalorprecordialpainforseveralhoursordays,whileirritability,sweating,frequentdeathflu,deliverydescendingarteryinfarctionblock.hospitalduringthe18patients,15casesmanifestedasnausea,vomiting,abdominalpain,accompaniedbyarrhythmiaearly,twocasesofsevereventricularfibrillation,cardiogenicshock,patientspresentwithirritability,pale,rapidshallowbreathing,threadyandweakpulse,clammyskin,sweati...

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